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Table of contents
Notes
1.
Scrap
1.1.
Sympathomimetics
1.1.1.
Adrenaline
1.1.2.
Noradrenaline
1.1.3.
Dopamine
1.1.4.
Phenylephrine
1.1.5.
Isoproterenol
1.1.6.
Dobutamine
1.1.7.
Dopexamine
1.1.8.
Salbutamol
1.1.9.
Ephedrine
1.1.10.
Metaraminol
1.1.11.
Phosphodiesterase inhibitor
1.2.
Drugs affecting coagulation
1.2.1.
Antiplatelet
1.2.2.
Anticoagulants
1.2.2.1.
Heparin
1.2.2.1.1.
Protamine
1.2.2.2.
Low-molecular-weight heparins (LMWH)
1.2.2.3.
Oral anticoagulants
1.3.
Adrenergic antagonists
1.3.1.
Prazosin
1.4.
Antibiotics
1.4.1.
Beta-lactam
1.4.1.1.
Penicillin
1.4.1.2.
Cephalosporins
1.4.1.3.
Carbapenems
1.4.2.
Macrolides
2.
Statistics
2.1.
Data types
2.2.
Descriptive statistics
2.3.
Principles of probability and inference
2.4.
Research design
3.
Pharmacology
3.1.
Pharmacology principles
3.1.1.
Pharmacokinetics
3.1.1.1.
Drug passage across cell membrane
3.1.1.2.
Absorption
3.1.1.3.
Distribution of drugs
3.1.1.3.1.
Volume of distribution
3.1.1.3.2.
Ionisation
3.1.1.3.3.
Protein-binding
3.1.1.4.
Clearance
3.1.1.5.
Drug metabolism
3.1.1.6.
Half-time and half-life
3.1.1.7.
Compartment model
3.1.2.
Pharmacodynamics
3.1.2.1.
Stereochemistry
3.1.2.2.
Mechanism of drug actions
3.1.2.2.1.
Receptors
3.1.2.2.1.1.
NMDA receptors
3.1.2.2.1.2.
G-protein system
3.1.2.2.1.3.
Receptor occupancy theory
3.1.2.3.
Dose-response relationship
3.1.2.3.1.
Drug response and tolerance
3.1.2.3.2.
Altered drug response
3.1.3.
Pharmacogenetics
3.1.3.1.
Porphyria
3.1.3.2.
Atypical plasma cholinesterase
3.1.3.3.
Malignant hyperthermia
3.1.3.3.1.
Dantrolene
3.1.4.
Phases of clinical trials
3.1.5.
Variability in drug response
3.2.
Inhalational anaesthetic agents
3.2.1.
Pharmacokinetics of inhalational anaesthetics
3.2.2.
Pharmacodynamics of inhalational anaesthetics
3.2.2.1.
Minimal alveolar concentration
3.2.2.2.
Mechanism of immobility by inhalational anaesthetics
3.2.2.3.
Mechanism of anaesthesia-induced unconsciousness
3.2.3.
Physical properties of inhalational anaesthetics
3.2.4.
Individual inhalational anaesthetic agents
3.2.4.1.
Ideal inhaled anaesthetic agent
3.2.4.2.
Halothane
3.2.4.3.
Enflurane
3.2.4.4.
Isoflurane
3.2.4.5.
Desflurane
3.2.4.6.
Sevoflurane
3.2.4.7.
Nitrous oxide
3.2.4.8.
Xenon
3.2.5.
Comparisons of inhalational agents
3.2.5.1.
CNS effects of inhalational anaesthetic agents
3.2.5.2.
CVS effects of inhalational anaesthetic agents
3.2.5.3.
Respiratory effects of inhalational anaesthetic agents
3.2.5.4.
Hepatic effects of inhalational anaesthetic agents
3.2.5.5.
Renal effects of inhalational anaesthetic agents
3.2.5.6.
Other effects of inhalational anaesthetic agents
3.2.5.7.
Metabolism of inhalational anaesthetic agents
3.2.5.8.
Carbon monoxide toxicity with inhalational anaesthetic agents
3.2.6.
CO2 absorbent
3.3.
IV anaesthetic agents
3.3.1.
Ideal IV anaesthetic agent
3.3.2.
Propofol
3.3.3.
Etomidate
3.3.4.
Ketamine
3.3.5.
Barbiturate
3.3.6.
Benzodiazepine
3.3.6.1.
Midazolam
3.3.6.2.
Diazepam
3.3.6.3.
Flumazenil
3.4.
Local anaesthetics
3.4.1.
Pharmacodynamics of local anaesthetics
3.4.2.
Pharmacokinetics of local anaesthetics
3.4.3.
Clinical uses of local anaesthetics
3.4.4.
Cocaine
3.4.5.
Lignocaine
3.4.6.
Eutectic mixture of local anaesthetics (EMLA)
3.5.
Opioids
3.5.1.
Neuraxial Opioids
3.5.2.
Opioid receptors
3.5.3.
Opioid agonists
3.5.3.1.
Pharmacodynamics of opioids
3.5.3.2.
Pharmacokinetics of opioids
3.5.3.3.
Morphine
3.5.3.3.1.
Morphine-6-glucuronide
3.5.3.4.
Pethidine
3.5.3.5.
Fentanyl
3.5.3.6.
Sufentanil
3.5.3.7.
Alfentanil
3.5.3.8.
Remifentanil
3.5.3.9.
Other opioid agonists
3.5.3.9.1.
Codeine
3.5.3.9.2.
Oxycodone
3.5.3.9.3.
Methadone
3.5.3.9.4.
Propoxyphene
3.5.3.9.5.
Tramadol
3.5.3.10.
Opioid dose conversion
3.5.4.
Opioid agonist-antagonists
3.5.4.1.
Pentazocine
3.5.4.2.
Buprenorphine
3.5.5.
Opioid antagonists
3.5.5.1.
Naloxone
3.5.5.2.
Naltrexone
3.5.5.3.
Nalmefene
3.5.5.4.
Methylnaltrexone
3.6.
NSAIDs
3.6.1.
COX-2 inhibitors
3.6.2.
Nonspecific NSAIDs
3.6.2.1.
Aspirin
3.6.2.2.
Paracetamol
3.7.
Neuromuscular blocking drugs
3.7.1.
Depolarising NMBDs
3.7.1.1.
Suxamethonium
3.7.2.
Non-depolarising NMBDs
3.7.2.1.
Long-acting nondepolarising NMBDs
3.7.2.1.1.
Pancuronium
3.7.2.2.
Intermediate-acting nondepolarising NMBDs
3.7.2.2.1.
Atracurium
3.7.2.2.2.
Cisatracurium
3.7.2.2.3.
Vecuronium
3.7.2.2.4.
Rocuronium
3.7.2.3.
Short-acting nondepolarising NMBDs
3.7.2.3.1.
Mivacurium
3.7.3.
Neuromuscular blockade
3.7.4.
Neuromuscular monitoring
3.8.
Acetylcholine-related drugs
3.8.1.
Anticholinesterase
3.8.2.
Cholinergic agonists
3.8.3.
Anticholinergic drugs
3.9.
Other Drugs
3.9.1.
Dextromethorphan
3.9.2.
Steroid
3.10.
Pharmacology tables
4.
Physiology
4.1.
Acid and Base
4.1.1.
Acid-base ionisation
4.1.2.
pH and pKa
4.2.
Neurophysiology
4.2.1.
Nausea and vomiting
4.2.2.
Circumventricular organs
4.2.3.
Acetylcholine
4.2.3.1.
Acetylcholine receptors
4.2.3.1.1.
Nicotinic acetylcholine receptors
4.2.3.1.2.
Muscarinic acetylcholine receptors
4.2.4.
Neuromuscular junction
4.2.5.
Gamma-aminobutyric acid (GABA)
4.2.5.1.
GABA receptors
4.2.6.
Glycine
4.3.
GIT physiology
4.3.1.
Stomach
4.4.
Haematology
4.4.1.
Haemostasis
4.4.1.1.
Clot formation
4.4.1.1.1.
Platelet plug formation
4.4.1.1.2.
Coagulation cascade
4.4.1.1.3.
Control of clot formation
4.4.1.2.
Fibrinolysis
4.4.1.3.
Coagulation factors
4.4.1.4.
Vitamin K
4.5.
Renal physiology
4.5.1.
Renal clearance
4.6.
Esterases
4.7.
Fluids
4.7.1.
Fluid compartments
4.7.2.
Extracellular fluid
4.8.
General physiology
4.8.1.
Osmolality and tonicity
4.8.1.1.
Colligative properties
5.
Clinical measurements
5.1.
Basic physics principles
5.1.1.
Diffusion
6.
Anatomy
6.1.
Spinal anatomy
7.
Anaesthetics
7.1.
Anaesthetic complications
7.2.
Entropy
7.3.
Serotonin syndrome
7.4.
Anaphylaxis
7.4.1.
Anaphylaxis vs anaphylactoid reaction
7.4.2.
Anaphylaxis treatment
8.
Others
8.3.
Abbreviations
9.
SAQ Pharmacology
9.1.
Pharmacodynamics SAQs
9.2.
Pharmacokinetics SAQs
9.3.
Other general pharmacology SAQs
9.4.
Inhalational anaesthetics SAQs
9.5.
IV anaesthetics SAQs
9.6.
Opioids SAQs
9.7.
NMBDs SAQs
Table of contents
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Index
Notes.html
Part of study notes by
LD99
Revised at 03/18/2007 23:29
Version 7